WEBVTT

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Repair begins with a biological response.

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Establishing that a treatment improves
recovery

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takes a different kind of evidence.

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BPC-157 is an experimental peptide

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made of fifteen amino acids.

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Laboratory and animal studies can suggest
mechanisms.

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They cannot, by themselves, establish that

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an injured person will recover faster.

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In a 2021 report, Lee and Padgett reviewed

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seventeen knee-pain patients and reached
sixteen for follow-up.

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Twelve had received BPC-157 alone;

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eleven reported significant pain
improvement.

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Four others received it with thymosin beta
four.

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Those reports came from an uncontrolled
study.

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Less pain does not demonstrate that tendon
or cartilage has healed.

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The digestive tract is another major
research focus.

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In rat experiments, BPC-157 reduced
experimentally induced stomach

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ulcers and improved healing in some fistula
models.

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These are encouraging animal findings.

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Human evidence includes a 2005 ulcerative-
colitis

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trial reported in a conference abstract.

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Fifty-three people were randomized.

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Disease-activity scores improved in both
groups.

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The between-group estimate was too
uncertain

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to establish a benefit over placebo.

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The FDA concluded that the available data
were

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inadequate to establish efficacy and safety
for that use.

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Gastrointestinal promise is a research
lead;

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reliable treatment success in people
remains unproven.

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The next step is a defined preparation, a
fair comparison,

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and consistent measures of symptoms,
function, and tissue change.

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Safety also needs adequate follow-up.

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World Peptide Foundation.

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Peptides in 2.

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Every experience matters.
